What can polyclonal B cell activation induce?

Polyclonal B cell activation is a process where multiple B cell clones are stimulated, leading to the production of antibodies against various antigens. This activation can occur due to various factors, such as exposure to certain pathogens, cytokines, or other immune stimuli that trigger B cell proliferation and differentiation.

One significant outcome of polyclonal B cell activation is the production of autoantibodies. Autoantibodies are antibodies that mistakenly target and react with a person's own tissues or components, which can contribute to autoimmune disorders. In scenarios where polyclonal activation occurs, the diverse range of B cell responses can lead to the generation of these autoantibodies, potentially causing harm to the body.

The other options reflect important immune functions but are not direct consequences of polyclonal B cell activation. Increased T cell activity is more associated with T cell activation and interaction with antigen-presenting cells rather than solely relying on B cells. Enhanced phagocytosis primarily involves macrophages and neutrophils responding to pathogens but is separate from the function of B cells and their antibody production. Antigen presentation is specifically a function of professional antigen-presenting cells, like dendritic cells, which present antigens to T cells, rather than a direct result of B cell